英国布里斯托大学联合伦敦大学学院及巴西圣保罗联邦大学的研究团队在《自主神经科学》期刊上发表最新发现，证实中等强度的有氧运动不仅增强心脏功能，还会对控制心脏的星状神经节产生显著的结构性重塑 1。该研究基于大鼠模型显示，经过10周的有氧训练后，心脏神经系统的改变呈现出明显的左右不对称性：右侧神经节的神经元数量约为左侧的四倍，而左侧神经元体积近乎翻倍，右侧则略微缩小 1。这一成果发表于2026年10月26日，为理解运动对心脏生理的影响提供了新的科学依据 1。
主要作者Dr. Augusto Coppi指出，这些神经簇如同心脏的调光开关，而运动以特定于侧别的方式对其进行了重塑 1。研究人员认为，这一发现有望优化针对心律失常、心绞痛以及“破碎之心”综合征等疾病的临床治疗方案，特别是涉及神经阻滞或去神经手术等领域 1。
Researchers from the University of Bristol, in collaboration with teams from University College London and the Federal University of São Paulo, have discovered that moderate-intensity aerobic exercise does more than simply strengthen cardiac function; it fundamentally restructures the nervous system controls governing the heart. 1 The study, published in the journal Autonomic Neuroscience on October 26, 2026, utilized a rat model to demonstrate that ten weeks of aerobic training induced specific remodeling in the stellate ganglia, resulting in significant asymmetrical changes between the left and right sides of the body. 1
The findings revealed that the number of neurons in the right-sided ganglia was approximately four times higher than those on the left side. 1 In contrast, the volume of neurons on the left side nearly doubled, while those on the right experienced a slight reduction in size. 1 Dr. Augusto Coppi, a lead author of the study, described these neural clusters as "dimmer switches" for the heart, noting that exercise reshapes them in a side-specific manner. 1 This insight into asymmetric neural adaptation offers new scientific grounds for developing more precise therapeutic interventions for conditions such as arrhythmia, angina, and broken heart syndrome, potentially optimizing treatments like nerve blocks or denervation surgeries. 1
评论
还没有评论，欢迎留下第一条。